Author

M. E. McNehlan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Research Focus:host-pathogen & immune response· 1 Organism:mouse· 1

Frequent Co-Authors

A. Smirnov· 1 shared C. L. Stallings· 1 shared C. S. Chowdhury· 1 shared D. Kreamalmeyer· 1 shared N. Dubey· 1 shared R. L. Kinsella· 1 shared R. Woodson· 1 shared S. K. Naik· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperImmunology

A. Smirnov

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1 paper

Recent·Most cited

Type I IFN signaling in the absence of IRGM1 promotes M. tuberculosis replication in immune cells by suppressing T cell responses

S. K. Naik, M. E. McNehlan, Y. Mreyoud, R. L. Kinsella, A. Smirnov, C. S. Chowdhury, S. R. McKee, N. Dubey, R. Woodson, D. Kreamalmeyer, C. L. Stallings

This study shows that IRGM1 deletion increases type I IFN signaling, which suppresses T cell responses and promotes M. tuberculosis replication in immune cells.

bioRxiv · Oct 3, 2023 · PreprintImmunology