Author

M. Eliezeck

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 1 Organism:mouse· 1 Assay:proteomics· 1 Approach:statistical model· 1

Frequent Co-Authors

B. d. L. Sanches· 1 shared D. B. Peruchetti· 1 shared F. C. S. Nogueira· 1 shared I. C. de Jesus· 1 shared J. A. M. Evaristo· 1 shared J. Rezende-Ribeiro· 1 shared J. S. Cruz· 1 shared K. H. Sampaio· 1 shared

Activity by Year

2024-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperPhysiology

B. d. L. Sanches

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1 paper

Recent·Most cited

Evidence that the monoamine oxidase B (MAO-B) plays a central role in the inotropic dysfunction induced by genetic deletion of the Mas-related-G protein-coupled receptor D (MrgD) in mice

L. Rodrigues-Ribeiro, J. Rezende-Ribeiro, S. Scalzo, M. L. Dias-Pinto, B. d. L. Sanches, M. Eliezeck, I. C. de Jesus, J. A. M. Evaristo, K. H. Sampaio, D. B. Peruchetti, J. S. Cruz, F. C. S. Nogueira, M. J. Campagnole-Santos, S. Guatimosim, R. A. S. Santos, T. Verano-Braga

This study identifies MAO-B hyperactivation as a key contributor to cardiac dysfunction in MrgD-KO mice, showing that MAO-B inhibition reverses contractile defects.

bioRxiv · Mar 27, 2024 · PreprintPhysiology