S. Guatimosim

Evidence that the monoamine oxidase B (MAO-B) plays a central role in the inotropic dysfunction induced by genetic deletion of the Mas-related-G protein-coupled receptor D (MrgD) in mice

This study identifies MAO-B hyperactivation as a key contributor to cardiac dysfunction in MrgD-KO mice, showing that MAO-B inhibition reverses contractile defects.

The kallikrein-kinin system is falling into pieces: bradykinin fragments are biological active peptides

This study investigates the biological activity of bradykinin fragments and finds they induce nitric oxide production and vasorelaxation independently of classical kinin receptors.