Author

T. Verano-Braga

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Pharmacology & Toxicology· 2 Physiology· 2

Signature Assays, Organisms & Methods

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Organism:mouse· 4 Assay:proteomics· 4 Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:host-pathogen & immune response· 1 Organism:rat· 1

Frequent Co-Authors

L. Rodrigues-Ribeiro· 3 shared G. B. Menezes· 2 shared I. M. Souza-Silva· 2 shared R. A. S. Santos· 2 shared S. Guatimosim· 2 shared S. Scalzo· 2 shared A. A. Peluso· 1 shared A. B. Diniz· 1 shared

Activity by Year

2020-2024 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 26 mutual co-author links

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Curved chords = mutual co-authorship

3 shared papersPhysiology

L. Rodrigues-Ribeiro

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Angiotensin-(1-5) is a Potent Endogenous Angiotensin AT2-Receptor Agonist

I. M. Souza-Silva, A. A. Peluso, K. Elsaafien, A. L. Nazarova, K. B. Assersen, L. Rodrigues-Ribeiro, M. Mohammed, A. F. Rodrigues, A. Nawrocki, L. A. Johansen, P. Jensen, A. D. de Kloet, E. G. Krause, M. DelBorgo, I. Maslov, , U. M. Steckelings

This study identifies angiotensin-(1-5) as a potent endogenous agonist of the AT2-receptor, showing its ability to increase nitric oxide release and lower blood pressure through AT2R-mediated mechanisms.

bioRxiv · Apr 5, 2024 · PreprintPharmacology & Toxicology

Evidence that the monoamine oxidase B (MAO-B) plays a central role in the inotropic dysfunction induced by genetic deletion of the Mas-related-G protein-coupled receptor D (MrgD) in mice

L. Rodrigues-Ribeiro, J. Rezende-Ribeiro, S. Scalzo, M. L. Dias-Pinto, B. d. L. Sanches, M. Eliezeck, I. C. de Jesus, J. A. M. Evaristo, K. H. Sampaio, D. B. Peruchetti, J. S. Cruz, F. C. S. Nogueira, M. J. Campagnole-Santos, S. Guatimosim, R. A. S. Santos, T. Verano-Braga

This study identifies MAO-B hyperactivation as a key contributor to cardiac dysfunction in MrgD-KO mice, showing that MAO-B inhibition reverses contractile defects.

bioRxiv · Mar 27, 2024 · PreprintPhysiology

Vagus nerve mediated liver-brain axis is a major regulator of the metabolic landscape in the liver

C. F. Brito, R. C. Fonseca, L. Rodrigues-Ribeiro, B. F. Vaz, G. S. Tofani, A. B. Diniz, P. Fernandes, N. A. Nunes, R. M. Pessoa, A. C. Oliveira, V. N. Cardoso, S. O. Fernandes, M. P. Oliveira, J. Alvarez-Leite, G. B. Menezes, , A. G. Oliveira

This study investigates the role of the vagus nerve in regulating liver metabolism, finding that its ablation alters hepatic proteome and promotes lipid accumulation and steatosis.

bioRxiv · Nov 16, 2023Physiology

The kallikrein-kinin system is falling into pieces: bradykinin fragments are biological active peptides

I. M. Souza-Silva, C. A. de Paula, A. K. Santos, V. L. S. Oliveira, I. D. Rocha, M. Antunes, L. P. B. Cordeiro, V. P. Teixeira, S. Scalzo, F. A. Amaral, J. M. Resende, M. A. P. Fontes, G. B. Menezes, S. Guatimosim, R. Santos, T. Verano-Braga

This study investigates the biological activity of bradykinin fragments and finds they induce nitric oxide production and vasorelaxation independently of classical kinin receptors.

bioRxiv · Sep 14, 2020 · PreprintPharmacology & Toxicology