Author

M. Mioc

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:differential expression· 1 Organism:human· 1 Approach:mechanistic / dynamical model· 1

Frequent Co-Authors

M. Kralj· 2 shared M. Marjanovic· 2 shared A.-M. M. Drazic· 1 shared B. Minofar· 1 shared D. Bonhenry· 1 shared F. Klicek· 1 shared G. Lauc· 1 shared J. Gabrilo· 1 shared

Activity by Year

2022-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 30 mutual co-author links

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2 shared papersCancer Biology

M. Kralj

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Targeting plasma membrane and mitochondrial instability in breast cancer cells and breast epithelial to mesenchymal transition-model cells by adamantyl diaza-crown ether ZG613

K. Ester, M. Mioc, P. Spurny, D. Bonhenry, M. Marjanovic, L. Uzelac, J. Gabrilo, T. Sumanovac, W. Schreibmayer, K. Majerski, B. Minofar, J. Ludwig, M. Kralj

This study evaluates ZG613's ability to disrupt plasma membranes and mitochondria in breast cancer and EMT-model cells, showing reduced tumor growth in vivo.

bioRxiv · Sep 30, 2022 · PreprintCancer Biology

Salinomycin disturbs Golgi apparatus function and specifically affects cells in epithelial-to-mesenchymal transition

M. Marjanovic, A.-M. M. Drazic, M. Mioc, F. Klicek, M. Novokmet, G. Lauc, M. Kralj

This study shows that salinomycin disrupts Golgi apparatus function and specifically harms cells undergoing epithelial-to-mesenchymal transition.

bioRxiv · Aug 31, 2022Cancer Biology