Author

M. Slak Rupnik

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2 Biophysics· 1

Signature Assays, Organisms & Methods

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Research Focus:cell signaling & trafficking· 2 Organism:mouse· 2 Research Focus:cell-cell communication· 1 Approach:deep learning· 1 Assay:electrophysiology· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:imaging· 1

Frequent Co-Authors

A. Stozer· 3 shared J. Dolensek· 3 shared M. Skelin Klemen· 3 shared D. Korosak· 2 shared V. Pohorec· 2 shared B. Podobnik· 1 shared C. Evans-Molina· 1 shared J. D. Johnson· 1 shared

Activity by Year

2019-2021 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 40 mutual co-author links

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3 shared papersPhysiology

A. Stozer

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

High resolution analysis of the cytosolic Ca2+ events in beta cell collectives in situ

S. Postic, S. Sarikas, J. Pfabe, V. Pohorec, L. Krizancic Bombek, N. Sluga, M. Skelin Klemen, J. Dolensek, D. Korosak, A. Stozer, C. Evans-Molina, J. D. Johnson, M. Slak Rupnik

This study measures Ca2+ dynamics in beta cell collectives using high-resolution imaging and a new computational framework, revealing three modes of Ca2+ event durations and the role of intracellular Ca2+ receptors.

bioRxiv · Apr 14, 2021Physiology

Glucose-dependent activation, activity, and deactivation of beta cell networks in acute mouse pancreas tissue slices

A. Stozer, M. Skelin Klemen, M. Gosak, L. Krizancic-Bombek, V. Pohorec, M. Slak Rupnik, J. Dolensek

This study investigates glucose-dependent changes in beta cell network activity in mouse pancreas tissue slices, revealing heterogeneity and functional connectivity patterns during activation, activity, and deactivation.

bioRxiv · Mar 11, 2020Physiology

β-cells operate collectively to help maintain glucose homeostasis

B. Podobnik, D. Korosak, M. Skelin Klemen, A. Stozer, J. Dolensek, M. Slak Rupnik, P. C. Ivanov, P. Holme, M. Jusup

The paper demonstrates that beta cells use collective communication to maintain glucose homeostasis through gap-junctional signaling and paracrine interactions, supported by a dynamical network model and empirical data analysis.

bioRxiv · Dec 1, 2019Biophysics