Author

Marc Lalande

University of Connecticut

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Genetics· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Christopher E. StoddardiD· 1 shared Clarisse OrniackiiD· 1 shared Clémence M Dupont-Thibert· 1 shared Dea GorkaiD· 1 shared Heather R. Glatt-DeeleyiD· 1 shared Justin L. CotneyiD· 1 shared Leann J Crandall· 1 shared Maéva LangouëtiD· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperGenetics

Christopher E. StoddardORCID

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

Specific ZNF274 binding interference at SNORD116 activates the maternal transcripts in Prader-Willi syndrome neurons

Maéva Langouët, Dea Gorka, Clarisse Orniacki, Clémence M Dupont-Thibert, Michael S Chung, Heather R. Glatt-Deeley, Noélle D. Germain, Leann J Crandall, Justin L. Cotney, Christopher E. Stoddard, Marc Lalande, Stormy J. Chamberlain

The paper shows that disrupting ZNF274 binding at SNORD116 activates its maternal allele in PWS neurons without altering ZNF274 levels, suggesting a therapeutic strategy for Prader-Willi syndrome.

Human Molecular Genetics · Sep 18, 2020Peer-reviewedGenetics