Author

Maéva Langouët

University of ConnecticutORCID 0000-0001-7176-0427Unclaimed ORCID profile · follow to get notified of new preprints

Additional preprints under this name without ORCID

Maéva Langouët1 paper · Institute for Research in Immunology and Cancer

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Assay:bulk RNA-seq· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

Christopher E. StoddardiD· 1 shared Clarisse OrniackiiD· 1 shared Clémence M Dupont-Thibert· 1 shared Dea GorkaiD· 1 shared Heather R. Glatt-DeeleyiD· 1 shared Justin L. CotneyiD· 1 shared Leann J Crandall· 1 shared Marc Lalande· 1 shared

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2020-2020 · peak 1/yr

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Christopher E. StoddardORCID

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1 paper

Recent·Most cited

GeneticsHuman Molecular GeneticsPeer-reviewedSep 18, 2020

Specific ZNF274 binding interference at SNORD116 activates the maternal transcripts in Prader-Willi syndrome neurons

Maéva Langouët, Dea Gorka, Clarisse Orniacki…·Stormy J. Chamberlain

The paper shows that disrupting ZNF274 binding at SNORD116 activates its maternal allele in PWS neurons without altering ZNF274 levels, suggesting a therapeutic strategy for Prader-Willi syndrome.

19 citations

humanbulk RNA-seq

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