Author

Michael S Chung

University of Connecticut

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Genetics· 2

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Assay:bulk RNA-seq· 2 Research Focus:differential expression· 2 Organism:human· 2 Research Focus:benchmarking· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Approach:statistical model· 1

Frequent Co-Authors

Christopher E. StoddardiD· 2 shared Justin L. CotneyiD· 2 shared Clarisse OrniackiiD· 1 shared Clémence M Dupont-Thibert· 1 shared Dea GorkaiD· 1 shared Gordon CarmichaeliD· 1 shared Heather R. Glatt-DeeleyiD· 1 shared Leann J Crandall· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersGenetics

Christopher E. StoddardORCID

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2 papers

Recent·Most cited

Identifying key underlying regulatory networks and predicting targets of orphan C/D box SNORD116 snoRNAs in Prader-Willi syndrome

Rachel B. Gilmore, Yaling Liu, Christopher E. Stoddard, Michael S Chung, Gordon Carmichael, Justin L. Cotney

The paper identifies a gene regulatory network controlled by SNORD116 snoRNAs and predicts their targets in Prader-Willi syndrome using isogenic cell lines and computational analysis.

Nucleic Acids Research · Nov 22, 2024Peer-reviewedGenetics

Specific ZNF274 binding interference at SNORD116 activates the maternal transcripts in Prader-Willi syndrome neurons

Maéva Langouët, Dea Gorka, Clarisse Orniacki, Clémence M Dupont-Thibert, Michael S Chung, Heather R. Glatt-Deeley, Noélle D. Germain, Leann J Crandall, Justin L. Cotney, Christopher E. Stoddard, Marc Lalande, Stormy J. Chamberlain

The paper shows that disrupting ZNF274 binding at SNORD116 activates its maternal allele in PWS neurons without altering ZNF274 levels, suggesting a therapeutic strategy for Prader-Willi syndrome.

Human Molecular Genetics · Sep 18, 2020Peer-reviewedGenetics