Author

Mehrnaz Zarei

Duke University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 1 Evolutionary Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:Drosophila· 1 Approach:experimental assay· 1 Organism:human· 1 Assay:scRNA-seq· 1 Approach:statistical model· 1

Frequent Co-Authors

Amber Navarra· 1 shared Brenda Manzano‐Winkler· 1 shared Edwin S. IverseniD· 1 shared Hannah John· 1 shared Hojat BornaiD· 1 shared Iman HamidiD· 1 shared Katrina Focht· 1 shared Mayte Suárez‐FariñasiD· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperEvolutionary Biology

Amber Navarra

Mutual co-authors in cluster

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintSep 22, 2026

Platelet-programmed circulating tumor cells signal to monocytes through a candidate CD40LG-ITGA5:ITGB1 myeloid checkpoint axis in breast cancer

Samane Khoshbakht, Hojat Borna, Mehrnaz Zarei…·Mayte Suárez‐Fariñas

This study identifies platelet-programmed CTCs and their communication with monocytes via CD40LG-ITGA5:ITGB1, showing an inverse association with survival in breast cancer.

DOI 10.64898/2026.09.20.752972

humanscRNA-seq

𝕏 PostBluesky

Evolutionary BiologybioRxivPreprintOct 16, 2025

Transposable elements drive much of naturally occurring genetic lethality in Drosophila melanogaster

Sarah B. Marion, Katrina Focht, Iman Hamid…·Mohamed A. F. Noor

This study identifies transposable element insertions as a major cause of recessive lethality in Drosophila melanogaster, suggesting they drive lethal mutation frequencies through repeated invasions and suppression.

DOI 10.1101/2025.10.16.682755

Drosophilaexperimental assay

𝕏 PostBluesky