Author

N. Liske

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Assay:DNA methylation· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Al-Fatlawi· 1 shared A. F. Stewart· 1 shared A. Gupta· 1 shared A. Kranz· 1 shared A. R. Poetsch· 1 shared C. Choudhary· 1 shared D. C. Torres· 1 shared D. Severinov· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperCell Biology

A. Al-Fatlawi

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1 paper

Recent·Most cited

The BOD1L subunit of the SETD1A complex sustains the expression of DNA damage repair genes despite restraining H3K4 trimethylation

G. Ciotta, S. Singh, A. Kranz, A. Gupta, D. C. Torres, J. Fu, R. Choudhury, N. Liske, W. K. Chu, C. Choudhary, L. Gahurova, D. Severinov, A. Al-Fatlawi, M. Schroeder, R. Aasland, , A. F. Stewart

The paper shows that BOD1L in the SETD1A complex maintains DNA repair gene expression despite reducing H3K4 trimethylation, and its loss causes DNA damage and cell death in ESCs.

bioRxiv · Apr 6, 2023 · PreprintCell Biology