Author

N. Messaddeq

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Bioengineering· 1 Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Organism:mouse· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

A. L. Charles· 1 shared A. Meyer· 1 shared B. Belbellaa· 1 shared B. GENY· 1 shared B. Keyes· 1 shared C. Spiegelhalter· 1 shared D. Bulavin· 1 shared D. Metzger· 1 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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1 shared paperImmunology

A. L. Charles

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Experimental myositis: an optimised version of C-protein-induced myositis

M. GIANNINI, D. Rovito, M. Oulad-Abdelghani, N. Messaddeq, L. Debrut, P. KESSLER, A. L. Charles, B. GENY, D. Metzger, G. Laverny, A. Meyer

This study optimises C-protein-induced myositis by immunising mice with a specific C-protein polypeptide, resulting in a reproducible and stable model of inflammatory myopathy with key pathological features.

bioRxiv · May 18, 2024Immunology

Fragmentation through cell-cell adhesion maintains senescent cell viability but promotes debris deposition

M. Durik, D. Sampaio Goncalves, T. Knauer Meyer, C. Spiegelhalter, N. Messaddeq, M. T. Seehawer, D. Bulavin, L. Zender, B. Keyes

This study identifies senescent-cell adhesion fragments (SCAFs) as a mechanism for senescent cell survival, but also for the release of damaged contents that may contribute to disease.

bioRxiv · Jan 11, 2023 · PreprintCell Biology

High levels of frataxin overexpression leads to mitochondrial and cardiac toxicity in mouse models

B. Belbellaa, L. Reutenauer, N. Messaddeq, L. Monassier, H. Puccio

This study shows that high frataxin overexpression in mouse models causes mitochondrial and cardiac toxicity, with toxicity emerging above 20-fold normal levels.

bioRxiv · Mar 31, 2020Bioengineering