Author

N. Vyas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Ecology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Approach:statistical model· 3 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:ecology & biodiversity· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Research Focus:host-pathogen & immune response· 1

Frequent Co-Authors

C. Mukhopadhyay· 2 shared M. Walsh· 2 shared S. Sawleshwarkar· 2 shared A. Aloysius· 1 shared A. Ghosh· 1 shared A. Pattanaik· 1 shared C. Webb· 1 shared D. Saxena· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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2 shared papersEcology

C. Mukhopadhyay

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

A preliminary description of the ecological characteristics of wild waterbird Japanese encephalitis virus hosts in high risk landscapes in India

M. Walsh, A. Pattanaik, N. Vyas, D. Saxena, C. Webb, S. Sawleshwarkar, C. Mukhopadhyay

This study identifies ecological profiles of Japanese encephalitis virus hosts in high-risk landscapes in India to guide wildlife surveillance.

bioRxiv · Jan 13, 2022 · PreprintEcology

Low mammalian species richness is associated with Kyasanur Forest disease outbreak risk in deforested landscapes in the Western Ghats, India

M. Walsh, R. Bhat, V. Nagarajan-Radha, P. Narayanan, N. Vyas, S. Sawleshwarkar, C. Mukhopadhyay

The paper assesses the relationship between mammalian species richness and Kyasanur Forest disease (KFDV) outbreak risk in deforested landscapes of the Western Ghats, finding that low species richness is linked to higher outbreak risk in areas with significant forest loss.

bioRxiv · Apr 8, 2021Ecology

The primary cilium dampens proliferative signaling and represses a G2/M transcriptional network in quiescent myoblasts

N. Venugopal, A. Ghosh, H. Gala, A. Aloysius, N. Vyas, J. Dhawan

This study shows that the primary cilium in quiescent myoblasts dampens proliferative signaling and represses G2/M transcriptional networks, contributing to cell cycle arrest.

bioRxiv · Nov 6, 2019Cell Biology