Author

Noah Brown

University of Virginia

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genomics· 2 Bioinformatics· 1

Signature Assays, Organisms & Methods

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Research Focus:variant calling· 3 Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Assay:whole-genome sequencing· 2 Research Focus:benchmarking· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

Jennifer L. GüleriD· 2 shared Abner Louissant· 1 shared Aishwarya GurumurthyiD· 1 shared Alec ChuiD· 1 shared Aleksander Łuniewski· 1 shared Anamarija M. PerryiD· 1 shared Anthony D. SchmittiD· 1 shared Aristotelis TsirigosiD· 1 shared

Activity by Year

2025-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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2 shared papersBioinformatics

Jennifer L. GülerORCID

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Author profile →

3 papers

Recent·Most cited

Hi-C for genome-wide detection of enhancer-hijacking rearrangements in routine lymphoid cancer biopsies

Jamin Wu, Alec Chu, Jang Cho, Misha Movahed-Ezazi, Kristyn Galbraith, Camila S. Fang, Yiying Yang, Chanel Schroff, Kristin L Sikkink, Michelle Perez-Arreola, Logan Van Meter, Savanna Gemus, Jon-Matthew Belton, Xue Song, Aishwarya Gurumurthy, , Russell J.H. Ryan

This study uses FFPE-compatible Hi-C to detect enhancer-hijacking rearrangements in lymphoid cancers, identifying clinically relevant drivers missed by standard methods.

Cell Genomics · Feb 20, 2026Peer-reviewedGenomics

SVCROWS: A User-Defined Tool for Interpreting Significant Structural Variants in Heterogeneous Datasets

Noah Brown, Charles Danis, Vazira Ahmedjanova, Jennifer L. Güler

SVCROWS improves structural variant interpretation by merging SVs with a size-weighted reciprocal overlap framework, maintaining accuracy and preserving rare genotypes in complex datasets.

Nucleic Acids Research · Dec 4, 2025Peer-reviewedBioinformatics

Replication stress increases de novo CNVs across the malaria parasite genome

Noah Brown, Aleksander Łuniewski, Xuanxuan Yu, Michelle D. Warthan, Shiwei Liu, Julia Zulawinska, Syed Ahmad, Nadia Prasad, Molly D. Congdon, Webster Santos, Feifei Xiao, Jennifer L. Güler

The paper shows replication stress increases de novo CNVs in Plasmodium falciparum, affecting genes involved in human infection.

Nucleic Acids Research · Dec 4, 2025Peer-reviewedGenomics