Author

P. Mulder

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Physiology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:development & morphogenesis· 2 Approach:experimental assay· 2 Research Focus:host-pathogen & immune response· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Organism:human· 1

Frequent Co-Authors

A. Dumesnil· 2 shared C. Heron· 2 shared E. Brakenhielm· 2 shared J.-B. Michel· 2 shared A. Lebon· 1 shared A. Ouvrard-Pascaud· 1 shared A. Zernecke· 1 shared C. Valentin· 1 shared

Activity by Year

2021-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 37 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPhysiology

A. Dumesnil

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Blocking Interleukin-1β transiently limits left ventricular dilation and reduces cardiac lymphangiogenesis during pressure-overload in mice

C. Heron, T. Lemarcis, O. Laguerre, M. Bundalo, C. Valentin, M. Valet, A. Dumesnil, J.-B. Michel, P. Mulder, A. Zernecke, V. Tardif San Martin, E. Brakenhielm

This study shows that blocking IL-1β early after pressure-overload reduces left ventricular dilation but not heart failure, while late blocking reduces lymphangiogenesis without worsening fibrosis.

bioRxiv · Apr 1, 2023 · PreprintPhysiology

Regulation and impact of cardiac lymphangiogenesis in pressure-overload-induced heart failure

C. Heron, A. Dumesnil, M. Houssari, S. Renet, A. Lebon, D. Godefroy, D. Schapman, O. Henri, G. Riou, L. Nicol, J.-P. Henry, M. Pieronne Deperrois, A. Ouvrard-Pascaud, R. Hagerling, H. Chiavelli, , E. Brakenhielm

This study investigates cardiac lymphangiogenesis in pressure-overload-induced heart failure, finding that it limits inflammation and fibrosis, delaying heart failure development.

bioRxiv · Apr 27, 2021Physiology