Author

P. S. Biswas

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:bulk RNA-seq· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1 Research Focus:gene regulatory network inference· 1

Frequent Co-Authors

D.-D. Li· 2 shared S. Gaffen· 2 shared Y. Li· 2 shared C. Jawale· 1 shared J. Kolls· 1 shared K. Ramani· 1 shared M. J. McGeachy· 1 shared R. Bechara· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersImmunology

D.-D. Li

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2 papers

Recent·Most cited

Arid5a mediates an IL-17-dependent pathway that drives autoimmunity but not antifungal host defense

T. C. Taylor, Y. Li, D.-D. Li, S. Majumder, M. J. McGeachy, P. S. Biswas, S. Gingras, S. Gaffen

This study shows Arid5a promotes IL-17-dependent autoimmunity but not antifungal immunity, suggesting targeted therapy could reduce autoimmune side effects.

bioRxiv · Feb 14, 2022Immunology

Antibody-induced glomerulonephritis is amplified by RTEC-intrinsic IL-17 signaling and restrained by IL-17-mediated induction of the endoribonuclease Regnase-1 (Zc3h12a)

D.-D. Li, R. Bechara, K. Ramani, C. Jawale, Y. Li, J. Kolls, S. Gaffen, P. S. Biswas

This study shows that IL-17 signaling in renal tubular epithelial cells drives antibody-induced glomerulonephritis, and Regnase-1 limits this inflammation by inhibiting IL-17 target genes.

bioRxiv · Jan 11, 2021 · PreprintImmunology