Author

R. Grencis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2 Research Focus:cell type annotation· 1 Assay:CITE-seq· 1 Research Focus:host-pathogen & immune response· 1 Assay:proteomics· 1

Frequent Co-Authors

A. Gallagher· 1 shared A. Howden· 1 shared A. Iseppon· 1 shared C. Moreira Heliodoro· 1 shared E. Stolarczyk· 1 shared G. Beattie· 1 shared G. Lord· 1 shared G. Panic· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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1 shared paperImmunology

A. Gallagher

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2 papers

Recent·Most cited

Amino acid availability acts as a metabolic rheostat to determine the magnitude of ILC2 responses

S. Hodge, M. Krauss, I. Kaymak, J. King, A. Howden, G. Panic, R. Grencis, J. Swann, L. Sinclair, M. Hepworth

This study shows that amino acid availability regulates ILC2 responses by affecting mTOR activation and expansion, highlighting nutritional cues in innate immunity.

bioRxiv · Jun 22, 2022Immunology

T-bet fate mapping identifies a novel ILC1-ILC2 subset in vivo

J.-H. SCHROEDER, N. Garrido-Mesa, T. Zabinski, A. Gallagher, L. Campbell, L. Roberts, E. Stolarczyk, G. Beattie, J. Lo, A. Iseppon, C. Moreira Heliodoro, R. Reis, R. Jenner, P. Lavender, J. Howard, , G. Lord

The paper identifies a novel ILC1-ILC2 subset using T-bet fate mapping and shows its dependence on T-bet and cytokine expression capabilities.

bioRxiv · Aug 21, 2020 · PreprintImmunology