Author

R. Moscona

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Approach:experimental assay· 2 Organism:human· 2 Approach:statistical model· 2 Assay:ATAC-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1

Frequent Co-Authors

A. Spatz· 2 shared E. Rubin· 2 shared M. Elchebly· 2 shared A. I. Papadakis· 1 shared A. Papadakis· 1 shared H. Wang· 1 shared L. C. van Kempen· 1 shared M. Redpath· 1 shared

Activity by Year

2019-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

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2 shared papersCancer Biology

A. Spatz

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2 papers

Recent·Most cited

BORIS/CTCFL-mediated chromatin accessibility alterations promote a pro-invasive transcriptional signature in melanoma cells

R. Moscona, S. Janssen, M. Elchebly, A. Papadakis, A. Spatz, E. Rubin

This study shows that BORIS/CTCFL alters chromatin accessibility in melanoma cells, promoting a pro-invasive transcriptional signature by upregulating invasion genes and downregulating tumor suppressors.

bioRxiv · Feb 11, 2022Cancer Biology

BORIS/CTCFL promotes a switch from a proliferative towards an invasive phenotype in melanoma cells

S. M. Janssen, R. Moscona, M. Elchebly, A. I. Papadakis, M. Redpath, H. Wang, E. Rubin, L. C. van Kempen, A. Spatz

This study shows that BORIS promotes a switch from a proliferative to an invasive phenotype in melanoma cells through transcriptional reprogramming.

bioRxiv · Jul 24, 2019Cancer Biology