Author

R. Truant

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 3 Scientific Communication & Education· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:cell signaling & trafficking· 2 Research Focus:benchmarking· 1 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:development & morphogenesis· 1 Research Focus:differential expression· 1

Frequent Co-Authors

C. E. Suart· 2 shared T. Maiuri· 2 shared A. M. Perez· 1 shared C. B. Bazan· 1 shared C. L. K. Hung· 1 shared C. Peng· 1 shared C. Suart· 1 shared I. Al-Ramahi· 1 shared

Activity by Year

2018-2021 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 16 mutual co-author links

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2 shared papersCell Biology

C. E. Suart

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Mod3D: A Low-Cost, Flexible Modular System of Live-Cell Microscopy Chambers and Holders

C. B. Bazan, S. Goss, C. Peng, N. Begeja, C. Suart, K. Neuman, R. Truant

The paper introduces a low-cost, modular live-cell microscopy system using 3D printing to reduce plastic waste and costs compared to commercial chambers.

bioRxiv · Oct 18, 2021 · PreprintCell Biology

Spinocerebellar Ataxia Type 1 protein Ataxin-1 is signalled to DNA damage by Ataxia Telangiectasia Mutated kinase

C. E. Suart, A. M. Perez, I. Al-Ramahi, T. Maiuri, J. Botas, R. Truant

This study shows that ataxin-1 localizes to DNA damage sites and is phosphorylated by ATM kinase, with polyglutamine expansions impairing this response, suggesting a link between DNA repair and SCA1 pathogenesis.

bioRxiv · Sep 18, 2020Cell Biology

Development of a knowledge translation platform for ataxia: Impact on readers and volunteer contributors

C. E. Suart, K. Graham, T. M. Nowlan Suart, R. Truant

This study evaluates the impact of SCAsource, a knowledge translation platform for ataxia research, on readers and volunteer contributors after one year.

bioRxiv · Feb 19, 2020Scientific Communication & Education

A Patient-Derived Cellular Model for Huntington Disease Reveals Phenotypes at Clinically Relevant CAG Lengths

C. L. K. Hung, T. Maiuri, L. E. Bowie, R. Gotesman, S. Son, M. Falcone, J. V. Giordano, V. Mattis, T. Lau, V. Kwan, V. Wheeler, J. Schertzer, K. Singh, R. Truant

This paper develops a patient-derived cellular model for Huntington Disease that maintains TP53 function and exhibits HD phenotypes at clinically relevant CAG lengths.

bioRxiv · Apr 12, 2018Cell Biology