Author

Ritobrata Bhattacharyya

Birla Institute of Technology and Science, Pilani

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 1 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:chromatin & epigenetic regulation· 2 Approach:experimental assay· 2 Assay:ChIP-seq· 1 Organism:human· 1 Organism:rat· 1

Frequent Co-Authors

Srinjoy ChakrabortyiD· 2 shared Syamantak MajumderiD· 2 shared Yash Tushar KatakiaiD· 2 shared Andrew AdvaniiD· 1 shared Gokula Narayanan· 1 shared Hariharan Jayakumar· 1 shared Nehal GuptaiD· 1 shared Nivedha Suresh· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 33 mutual co-author links

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2 shared papersBiochemistry

Srinjoy ChakrabortyORCID

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2 papers

Recent·Most cited

Splicing factor proline- and glutamine-rich (SFPQ) protein causes transcriptional repression of SNAIL to counteract TGF-β signaling

Niyati Pandya Thakkar, Hariharan Jayakumar, Shyam Kumar Ramakrishnan, Gokula Narayanan, Srinjoy Chakraborty, Ritobrata Bhattacharyya, Yash Tushar Katakia, Suzanne L. Advani, Andrew Advani, Syamantak Majumder

This study identifies SFPQ as a transcriptional repressor of Snail that counteracts TGF-β signaling by inhibiting Snail expression.

Biochimica et Biophysica Acta (BBA) - Gene Regulatory Mechanisms · Aug 19, 2026Peer-reviewedBiochemistry

DOT1L-AF10-mediated H3K79me3 promotes NF-kB p65-dependent inflammatory activation in endothelial cells

Yash Tushar Katakia, Ritobrata Bhattacharyya, Sushmitha Duddu, Nivedha Suresh, Srinjoy Chakraborty, Nehal Gupta, Suchit Chebolu, Praphulla Chandra Shukla, Syamantak Majumder

This study shows that DOT1L-mediated H3K79me3 promotes NF-κB p65-dependent inflammation in endothelial cells, suggesting it as a therapeutic target for atherogenesis.

bioRxiv · Mar 23, 2026 · PreprintCell Biology