DOT1L-AF10-mediated H3K79me3 promotes NF-kB p65-dependent inflammatory activation in endothelial cells
Yash Tushar Katakia, Ritobrata Bhattacharyya, Sushmitha Duddu…·Syamantak Majumder
This study shows that DOT1L-mediated H3K79me3 promotes NF-κB p65-dependent inflammation in endothelial cells, suggesting it as a therapeutic target for atherogenesis.