Author

S. A. Lynch

University College Cork

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genomics· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 1 Approach:experimental assay· 1 Organism:mouse· 1 Organism:non-human primate· 1 Research Focus:phylogenetics / evolution· 1 Approach:statistical model· 1 Assay:whole-genome sequencing· 1

Frequent Co-Authors

Andrés BlancoiD· 1 shared D. C. Hughes· 1 shared D. S. Waddell· 1 shared Inés Martínez SambadeiD· 1 shared J. R. Driscoll· 1 shared L. M. Baehr· 1 shared Manuel VeraiD· 1 shared Miguel HermidaiD· 1 shared

Activity by Year

2020-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 20 mutual co-author links

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1 shared paperGenomics

Andrés BlancoORCID

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2 papers

Recent·Most cited

Disentangling the role of chromosome inversions in the structure of flat oyster (Ostrea edulis): origin, adaptation and phylogeography

Inés Martínez Sambade, Vivian Hlordzi, Andrés Blanco, Miguel Hermida, Manuel Vera, Sarah C. Culloty, Tim P. Bean, S. A. Lynch, Paulino Martı́nez

The paper identifies and analyzes chromosomal inversions in the flat oyster, showing their role in adaptation and phylogeography through genomic and population studies.

bioRxiv · Jun 12, 2025 · PreprintGenomics

Identification and Characterization of Fbxl22, a novel skeletal muscle atrophy-promoting E3 ubiquitin ligase

D. C. Hughes, L. M. Baehr, J. R. Driscoll, S. A. Lynch, D. S. Waddell, S. C. Bodine

This study identifies Fbxl22 and its splice variant as E3 ubiquitin ligases involved in neurogenic muscle atrophy, showing their role in muscle atrophy and potential therapeutic relevance.

bioRxiv · Apr 24, 2020Physiology