Author

S. Alivernini

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Immunology· 2 Cell Biology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 3 Research Focus:differential expression· 3 Organism:human· 3 Approach:statistical model· 3 Approach:deep learning· 2 Assay:scRNA-seq· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1

Frequent Co-Authors

M. Kurowska-Stolarska· 3 shared B. Tolusso· 2 shared C. D. Buckley· 2 shared E. Gremese· 2 shared L. MacDonald· 2 shared A. Antonopoulos· 1 shared A. Bhalla· 1 shared A. Dell· 1 shared

Activity by Year

2020-2023 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 32 mutual co-author links

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3 shared papersImmunology

M. Kurowska-Stolarska

Mutual co-authors in cluster

Author profile →

3 papers

Recent·Most cited

Primed to resolve: A single cell atlas of the shoulder capsule reveals a cellular basis for resolving inflammatory fibrosis

M. T. Ng, R. Borst, H. Gacaferi, S. Davidson, C. C. Machado, I. Reekie, M. Attar, D. Windell, M. Kurowska-Stolarska, L. MacDonald, S. Alivernini, M. Garvilles, K. Jansen, A. Bhalla, A. Lee, , S. G. Dakin

This study identifies MERTK+ macrophages and fibroblasts involved in resolving fibrosis in frozen shoulder using single-cell analysis and suggests therapeutic strategies to enhance their interaction.

bioRxiv · Jan 16, 2023 · PreprintCell Biology

COVID-19 and Rheumatoid Arthritis share myeloid pathogenic and resolving pathways

L. MacDonald, T. D. Otto, A. Elmesmari, B. Tolusso, D. Somma, C. McSharry, E. Gremese, I. B. McInnes, S. Alivernini, M. Kurowska-Stolarska

This study identifies shared myeloid pathways between COVID-19 pneumonitis and RA synovitis, highlighting MerTK as a therapeutic target for both conditions.

bioRxiv · Jul 26, 2020 · PreprintImmunology

Loss of α2-6 sialylation promotes the transformation of synovial fibroblasts into a pro-inflammatory phenotype in Rheumatoid Arthritis.

Y. Wang, A. Khan, A. Antonopoulos, L. Bouche, C. D. Buckley, A. Filer, K. Raza, K. Ping-Li, B. Tolusso, E. Gremese, M. Kurowska-Stolarska, S. Alivernini, A. Dell, S. M. Haslam, M. A. Pineda

This study shows that reduced α2-6 sialylation in synovial fibroblasts contributes to a pro-inflammatory phenotype in RA by altering glycosylation pathways.

bioRxiv · Mar 8, 2020Immunology