The dysregulated IL-23/TH17 axis in endometriosis pathophysiology
D. J. Sisnett, K. B. Zutautas, J. E. Miller, H. Lingegowda, S. H. Ahn, A. McCallion, O. Bougie, B. A. Lessey, C. Tayade
This study investigates the role of the IL-23/TH17 axis in endometriosis by analyzing patient samples, cell lines, and a mouse model, finding dysregulated gene expression and increased IL-23 protein levels associated with disease severity.
bioRxiv · Dec 7, 2023Immunology