S. Rudaz

N -acetylglucosamine supplementation fails to bypass the critical acetylation of glucosamine-6-phosphate required for Toxoplasma gondii replication and invasion

This study shows that N-acetylglucosamine supplementation cannot restore UDP-GlcNAc levels or function in T. gondii when GNA1 is depleted, highlighting the critical role of GlcN6P acetylation in parasite replication and invasion.

Bacterial cell cycle control by citrate synthase independent of enzymatic activity

The paper identifies CitA, a citrate synthase in Caulobacter crescentus, as a checkpoint regulator of the G1-to-S transition independent of its enzymatic activity.