Author

Sabina Khan

University of California, Davis

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Developmental Biology· 1 Pathology· 1

Signature Assays, Organisms & Methods

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Organism:mouse· 2 Approach:statistical model· 2 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

Brian C. LeonardiD· 2 shared Jaegook LimiD· 2 shared Meher J. Khan· 2 shared Sangwan ParkiD· 2 shared Sara M. ThomasyiD· 2 shared Gabriella L. Donovan· 1 shared Hanna ShevalyeiD· 1 shared Hiroko IwashitaiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersDevelopmental Biology

Brian C. LeonardORCID

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2 papers

Recent·Most cited

Interplay between ferroptosis and guttae in an early-onset murine model of Fuchs endothelial corneal dystrophy (FECD)

Karin W. Handel, Jaegook Lim, Hiroko Iwashita, Sabina Khan, Hanna Shevalye, Sangwan Park, Nayeli Echeverria, Michelle Ferneding, Meher J. Khan, Karolina P. Roszak, Gabriella L. Donovan, 岩本真子, Jaeho Shim, Laura J. Young, Monica Ardon, , Sara M. Thomasy

This study shows that ferroptosis is linked to guttae formation in a murine model of Fuchs endothelial corneal dystrophy, providing a model for testing ferroptosis-targeted therapies.

bioRxiv · Jul 10, 2026 · PreprintDevelopmental Biology

TAZ (Wwtr1) deficiency leads to ER stress and mitochondrial dysfunction in a mouse model of Fuchs' endothelial corneal dystrophy

Sangwan Park, Raneesh Ramarapu, Jaegook Lim, Sabina Khan, Meher J. Khan, William R Stoehr, Brian C. Leonard, Sara M. Thomasy

This paper investigates TAZ deficiency in a mouse model of FECD, identifying ER stress, mitochondrial dysfunction, and altered ECM organization as key pathogenic mechanisms.

bioRxiv · Feb 19, 2026 · PreprintPathology