Author

Sandhini Saha

Regional Centre for Biotechnology

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2 Cancer Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Organism:human· 3 Research Focus:cancer & tumor microenvironment· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Assay:proteomics· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1

Frequent Co-Authors

Tushar Kanti MaitiiD· 4 shared Ankit BiswasiD· 3 shared Krishna Singh BishtiD· 2 shared Naman KharbandaiD· 2 shared Amrutha Nisthul A.· 1 shared Archana Praveen· 1 shared Arun (Geetha) ViswanathaniD· 1 shared Chenicheri Kizhakkeveettil KeerthanaiD· 1 shared

Activity by Year

2026-2026 · peak 4/yr

Co-Author Collaboration Network

10 top collaborators · 24 mutual co-author links

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4 shared papersBiochemistry

Tushar Kanti MaitiORCID

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

Integrated proteomic and phosphoproteomic profiling reveals mechanisms of Bisphenol-A induced placental toxicity

Ankit Biswas, Sandhini Saha, Naman Kharbanda, Tushar Kanti Maiti

This study identifies BPA-induced changes in placental phosphoproteome and kinase activity, revealing altered phosphorylation of c-JUN and GSK3 linked to placental toxicity and adverse pregnancy outcomes.

Chemical Research in Toxicology · Jul 13, 2026Peer-reviewedBiochemistry

Bisphenol-A mediated ubiquitinome alteration triggers PPAR-alpha ubiquitination, affecting trophoblast cell migration

Ankit Biswas, Sandhini Saha, Debapriyo Sarmadhikari, Krishna Singh Bisht, Shailendra Asthana, Tushar Kanti Maiti

This study identifies BPA-induced ubiquitinome changes in trophoblast cells, showing PPAR-alpha hyper-ubiquitination disrupts migration and may contribute to placental dysfunction.

bioRxiv · May 8, 2026 · PreprintBiochemistry

Uttroside B, a US FDA-designated Orphan drug, mitigates thedevelopment of hepatocellular carcinoma and its pulmonary metastasis via EGFR/ERK-mediated inhibition of SREBP-1 and STAT-3

Chenicheri Kizhakkeveettil Keerthana, Tennyson Prakash Rayginia, Kalimuthu Kalishwaralal, Sandhini Saha, Nair Hariprasad Haritha, Amrutha Nisthul A., Mundanattu Swetha, Sreekumar Usha Devi Aiswarya, Lekshmi R. Nath, Sanjay Suresh Varma, Arun (Geetha) Viswanathan, Somaraj Jannet, Shirly J, Jayasekharan S. Aparna, Archana Praveen, , Ruby John Anto

This study shows that Uttroside B inhibits hepatocellular carcinoma and its pulmonary metastasis by blocking the EGFR/ERK pathway and its downstream targets SREBP-1 and STAT-3.

Cell Death Discovery · Apr 16, 2026Peer-reviewedCancer Biology

Ferroptosis links α-synuclein pathology across brain and skeletal muscle in Parkinsons disease

Krishna Singh Bisht, Jaydeep Sharma, Naman Kharbanda, Ankit Biswas, Sandhini Saha, Sam Jacob Mathew, Tushar Kanti Maiti

This study identifies ferroptosis as a shared mechanism linking brain and skeletal muscle pathology in Parkinson's disease through α-synuclein-TFRC interactions.

bioRxiv · Apr 10, 2026 · PreprintNeuroscience