Author

Simo Ryhänen

University of Eastern Finland

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Neuroscience· 1

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Research Focus:differential expression· 1 Organism:human· 1 Organism:mouse· 1 Approach:statistical model· 1

Frequent Co-Authors

Annakaisa HaapasaloiD· 1 shared Benjam KemiläineniD· 1 shared Eija RahuneniD· 1 shared Heikki TanilaiD· 1 shared Helena SvobodováiD· 1 shared Henna MartiskaineniD· 1 shared Hennariikka Koivisto· 1 shared Jussi PihlajamäkiiD· 1 shared

Activity by Year

2020-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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1 shared paperNeuroscience

Annakaisa HaapasaloORCID

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1 paper

Recent·Most cited

Diabetic phenotype in mouse and humans with β-amyloid pathology reduces the number of microglia around β-amyloid plaques

Teemu Natunen, Henna Martiskainen, Mikael Marttinen, Sami Gabbouj, Hennariikka Koivisto, Susanna Kemppainen, Satu Kaipainen, Mari Takalo, Helena Svobodová, Luukas Leppänen, Benjam Kemiläinen, Simo Ryhänen, Teemu Kuulasmaa, Eija Rahunen, Sisko Juutinen, , Mikko Hiltunen

This study shows that a diabetic phenotype in mice and humans reduces microglial response to β-amyloid plaques, linking T2D and AD pathologies.

Molecular Neurodegeneration · Nov 9, 2020Peer-reviewedNeuroscience