Author

Susanna Kemppainen

University of Eastern FinlandORCID 0000-0001-5372-1918Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Research Focus:differential expression· 2 Organism:mouse· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell-cell communication· 1 Approach:experimental assay· 1 Research Focus:gene regulatory network inference· 1 Organism:human· 1

Frequent Co-Authors

Annakaisa HaapasaloiD· 2 shared Heikki TanilaiD· 2 shared Henna MartiskaineniD· 2 shared Mari TakaloiD· 2 shared Mikko HiltuneniD· 2 shared Petra M. Mäkinen· 2 shared Teemu KuulasmaaiD· 2 shared Teemu NatuneniD· 2 shared

Activity by Year

2020-2024 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

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2 shared papersNeuroscience

Annakaisa HaapasaloORCID

Mutual co-authors in cluster

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintSep 12, 2024

Protective variant in PLCgamma2 mitigates Alzheimer's disease associated pathology via enhancing beneficial microglia functions

Mari Takalo, Heli Jeskanen, Taisia Rõlova…·Mikko Hiltunen

This study shows that the PLCgamma2-P522R variant reduces beta-amyloid pathology and enhances microglial function in AD models, suggesting an anti-ageing mechanism.

DOI 10.1101/2024.09.12.612207

mouseproteomics

𝕏 PostBluesky

NeuroscienceMolecular NeurodegenerationPeer-reviewedNov 9, 2020

Diabetic phenotype in mouse and humans with β-amyloid pathology reduces the number of microglia around β-amyloid plaques

Teemu Natunen, Henna Martiskainen, Mikael Marttinen…·Mikko Hiltunen

This study shows that a diabetic phenotype in mice and humans reduces microglial response to β-amyloid plaques, linking T2D and AD pathologies.

35 citations

mousehuman

𝕏 PostBluesky