Author

T. Gudermann

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 2 Biochemistry· 1 Physiology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 4 Approach:experimental assay· 4 Research Focus:differential expression· 2 Organism:human· 2 Organism:mouse· 2 Assay:proteomics· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1

Frequent Co-Authors

A. Dietrich· 2 shared S. Rajan· 2 shared A. Breit· 1 shared A. Haupt· 1 shared A. Kollewe· 1 shared A. Nicke· 1 shared B. Fakler· 1 shared B. Koletzko· 1 shared

Activity by Year

2020-2022 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 13 mutual co-author links

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2 shared papersCell Biology

A. Dietrich

Mutual co-authors in cluster

Author profile →

4 papers

Recent·Most cited

An Inhibitory Function of TRPA1 Channels in TGF-β1-driven Fibroblast to Myofibroblast Differentiation

F. Geiger, S. Zeitlmayr, C. A. Staab-Weijnitz, S. Rajan, A. Breit, T. Gudermann, A. Dietrich

This study shows TRPA1 channels inhibit TGF-β1-driven fibroblast to myofibroblast differentiation and may protect against pulmonary fibrosis.

bioRxiv · Apr 12, 2022Cell Biology

The molecular appearance of native TRPM7 channel complexes identified by high-resolution proteomics

A. Kollewe, V. Chubanov, F. T. Tseung, A. Haupt, C. S. Müller, W. Bildl, S. Schulte, A. Nicke, B. Fakler, T. Gudermann

The paper identifies native TRPM7 channel complexes in rodent brain using proteomics and confirms their functional relevance through reconstitution experiments.

bioRxiv · Jul 9, 2021Biochemistry

The epoxyeicosatrienoic pathway is intact in endothelial and smooth muscle cells exposed to aldosterone excess

L. Brunnenkant, Y. Meng, J. Sun, J. Gonzalez Marques, B. Koletzko, M. Mederos y Schnitzler, T. Gudermann, T. A. Williams, F. Beuschlein, D. A. Heinrich, C. Adolf, M. Reincke, H. Schneider

This study investigates the effect of aldosterone excess on the epoxyeicosatrienoic acid (EET) pathway in endothelial and smooth muscle cells, finding no significant changes in gene expression or EET release.

bioRxiv · Feb 4, 2021 · PreprintPhysiology

Transient receptor vanilloid 4 (TRPV4) channels are essential for alveolar epithelial cell function

J. Weber, Y.-K. Chao, M. Kannler, G. Krasteva-Christ, S. Rajan, O. A. Yildirim, M. Brosien, J. Schredelseker, N. Weissmann, c. grimm, T. Gudermann, A. Dietrich

This study shows TRPV4 channels protect alveolar epithelial barriers by maintaining AQP-5 and pSP-C expression, preventing edema during ischemia-reperfusion.

JCI Insight · Oct 15, 2020Peer-reviewedCell Biology

T. Gudermann · henhao