Author

T. Sorlie

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Organism:human· 2 Approach:benchmark study· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:clustering· 1

Frequent Co-Authors

H. Bergholtz· 2 shared J. H. Norum· 2 shared A. Hurtado-Rodriguez· 1 shared A.-L. Borresen-Dale· 1 shared A.-M. Fosdahl· 1 shared C. Fougner· 1 shared E. Fiorito· 1 shared K. K. Sahlberg· 1 shared

Activity by Year

2019-2020 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersCancer Biology

H. Bergholtz

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Author profile →

2 papers

Recent·Most cited

Breast tumors escape endocrine therapy by ER-independent mechanisms triggered by the coordinated activities of HER2/HER3 and deacetylated FOXA1

S. Gilfillan, S. Wang, M. R. Katika, J. H. Norum, H. Bergholtz, E. Fiorito, S. Nordhagen, Y. Sharma, S. Singh, V. S. Somisety, A.-M. Fosdahl, S. Nord, O. Engebraaten, O. C. Lingjaerde, A.-L. Borresen-Dale, , A. Hurtado-Rodriguez

This study shows that HER2/HER3 signaling and FOXA1 deacetylation drive ER-independent growth in hormone-resistant breast tumors by altering FOXA1 chromatin binding and function.

bioRxiv · Sep 22, 2020 · PreprintCancer Biology

Re-definition of claudin-low as a breast cancer phenotype

C. Fougner, H. Bergholtz, J. H. Norum, T. Sorlie

This study re-defines claudin-low as a complex breast cancer phenotype by analyzing genomic, transcriptomic, and clinical features, revealing its heterogeneity and distinct characteristics.

bioRxiv · Sep 9, 2019Cancer Biology