Author

T. Zal

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Immunology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:scRNA-seq· 1

Frequent Co-Authors

B. Patel· 1 shared D. Kumar· 1 shared E. M. Park· 1 shared F. G. Giancotti· 1 shared F. Ma· 1 shared J. E. Pineda· 1 shared K. Clise-Dwyer· 1 shared L. M. Kahn· 1 shared

Activity by Year

2023-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 45 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperImmunology

B. Patel

Mutual co-authors in cluster

Author profile →

1 paper

Recent·Most cited

STAT3 protects HSCs from intrinsic interferon signaling and loss of long-term blood-forming activity

B. Patel, Y. Zhou, R. L. Babcock, F. Ma, M. A. Zal, D. Kumar, Y. B. Medik, L. M. Kahn, J. E. Pineda, E. M. Park, X. Tang, M. G. Raso, T. Zal, K. Clise-Dwyer, F. G. Giancotti, , S. S. Watowich

This study shows that STAT3 is essential for maintaining hematopoietic stem cell function by suppressing interferon signaling and myeloid skewing, and its loss leads to impaired hematopoietic activity.

bioRxiv · Feb 10, 2023 · PreprintImmunology