Author

Takeshi Nakamura

RIKEN Center for Brain SciencePrimary ORCID profile (0000-0003-2974-6837 · 2 papers)Unclaimed ORCID profile · follow to get notified of new preprints

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Same name, identified by ORCID

Takeshi NakamuraiD2 papers · Research Institute for Biomedical Sciences, Tokyo University of Science

Biology Disciplines

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Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Approach:experimental assay· 1 Organism:mouse· 1 Assay:proteomics· 1

Frequent Co-Authors

Atsushi MiyawakiiD· 1 shared Hideaki MizunoiD· 1 shared Ikuyo Inoue· 1 shared Katsuhiko Mikoshiba· 1 shared Kazuhiro YamakawaiD· 1 shared Kripamoy AguaniD· 1 shared Toshimitsu SuzukiiD· 1 shared

Activity by Year

2025-2025 · peak 1/yr

Co-Author Collaboration Network

7 top collaborators · 21 mutual co-author links

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1 shared paperPhysiology

Atsushi MiyawakiORCID

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1 paper

Recent·Most cited

Epilepsy protein myoclonin1 interacts with inositol 1,4,5-trisphosphate (IP3) receptor and reduces Ca2+ store in endoplasmic reticulum

Toshimitsu Suzuki, Kripamoy Aguan, Hideaki Mizuno, Takeshi Nakamura, Ikuyo Inoue, Katsuhiko Mikoshiba, Atsushi Miyawaki, Kazuhiro Yamakawa

This study identifies myoclonin1's interaction with IP3 receptors and PRKCSH, showing it modulates ER-Ca2+ homeostasis, which may contribute to epilepsy in EFHC1 mutation carriers.

Exploration of neuroscience · Jul 9, 2025Peer-reviewedPhysiology