Author

Timothy P. Rutkowski

Emory University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

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Genetics· 2

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Approach:experimental assay· 2 Organism:mouse· 2 Assay:metabolomics· 1

Frequent Co-Authors

David WeinshenkeriD· 2 shared Gary Jonathan BasselliD· 2 shared Jennifer Gladys MulleiD· 2 shared Michael P. EpsteiniD· 2 shared Rebecca M. PollakiD· 2 shared Ryan H. PurcelliD· 2 shared Stephen T. WarreniD· 2 shared Tamara CasparyiD· 2 shared

Activity by Year

2019-2020 · peak 1/yr

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10 top collaborators · 45 mutual co-author links

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2 shared papersGenetics

David WeinshenkerORCID

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2 papers

Recent·Most cited

Metabolic effects of the schizophrenia-associated 3q29 deletion are sex-specific and uncoupled from behavioral phenotypes

Rebecca M. Pollak, Ryan H. Purcell, Timothy P. Rutkowski, Tamika Malone, Kimberly J. Pachura, Gary Jonathan Bassell, Michael P. Epstein, Paul A. Dawson, Matthew Ryan Smith, Dean P. Jones, Michael E. Zwick, the Emory 3q29 Project, Stephen T. Warren, Tamara Caspary, David Weinshenker, Jennifer Gladys Mulle

This study shows that the 3q29 deletion causes sex-specific metabolic changes in mice, which are not linked to behavioral issues, and that a high-fat diet can improve weight in females without worsening behavior.

bioRxiv · Sep 18, 2020 · PreprintGenetics

Behavioral changes and growth deficits in a CRISPR engineered mouse model of the schizophrenia-associated 3q29 deletion

Timothy P. Rutkowski, Ryan H. Purcell, Rebecca M. Pollak, Stephanie M. Grewenow, Georgette M. Gafford, Tamika Malone, Uswa A. Khan, Jason P. Schroeder, Michael P. Epstein, Gary Jonathan Bassell, Stephen T. Warren, David Weinshenker, Tamara Caspary, Jennifer Gladys Mulle

This study creates a CRISPR-engineered mouse model of the 3q29 deletion, showing behavioral and growth deficits, and demonstrates that these effects are not solely due to Dlg1 haploinsufficiency.

Molecular Psychiatry · Apr 11, 2019Peer-reviewedGenetics