Author

Jason P. Schroeder

Emory University

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Genetics· 1 Molecular Biology· 1 Neuroscience· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Organism:mouse· 2 Research Focus:benchmarking· 1 Research Focus:drug response / drug discovery· 1 Organism:human· 1 Research Focus:protein design· 1 Research Focus:protein structure prediction· 1 Assay:proteomics· 1

Frequent Co-Authors

David WeinshenkeriD· 3 shared Alexia MarriottiD· 1 shared Anu KorukondaiD· 1 shared Brittany S. PateiD· 1 shared Carolina Montañez‐MirandaiD· 1 shared Christopher D. SchareriD· 1 shared Daniel J. LustbergiD· 1 shared Eric A. OrtlundiD· 1 shared

Activity by Year

2019-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 22 mutual co-author links

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3 shared papersNeuroscience

David WeinshenkerORCID

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3 papers

Recent·Most cited

Impact of early locus coeruleus lesions in the TgF344 Alzheimer's disease rat model

Alexia Marriott, Jason P. Schroeder, Anu Korukonda, Brittany S. Pate, Katharine E McCann, David Weinshenker, Michael A. Kelberman

This study investigates the impact of early locus coeruleus lesions in TgF344-AD rats and finds that LC lesions and AD genotype rarely interact to worsen deficits or neuropathology.

Alzheimer s & Dementia · Apr 1, 2026Peer-reviewedNeuroscience

Human genetic variants disrupt RGS14 nuclear shuttling and regulation of LTP in hippocampal neurons

Katherine E. Squires, Kyle J. Gerber, Matthew C. Tillman, Daniel J. Lustberg, Carolina Montañez‐Miranda, Meilan Zhao, Suneela Ramineni, Christopher D. Scharer, Ramendra N. Saha, Fengjue Shu, Jason P. Schroeder, Eric A. Ortlund, David Weinshenker, Serena M. Dudek, John R. Hepler

The paper identifies genetic variants in RGS14 that disrupt nuclear shuttling and impair LTP regulation in hippocampal neurons, with variant LR causing nuclear accumulation and loss of function.

Journal of Biological Chemistry · Nov 5, 2020Peer-reviewedMolecular Biology

Behavioral changes and growth deficits in a CRISPR engineered mouse model of the schizophrenia-associated 3q29 deletion

Timothy P. Rutkowski, Ryan H. Purcell, Rebecca M. Pollak, Stephanie M. Grewenow, Georgette M. Gafford, Tamika Malone, Uswa A. Khan, Jason P. Schroeder, Michael P. Epstein, Gary Jonathan Bassell, Stephen T. Warren, David Weinshenker, Tamara Caspary, Jennifer Gladys Mulle

This study creates a CRISPR-engineered mouse model of the 3q29 deletion, showing behavioral and growth deficits, and demonstrates that these effects are not solely due to Dlg1 haploinsufficiency.

Molecular Psychiatry · Apr 11, 2019Peer-reviewedGenetics