Author

Ujwal Subedi

Mayo Clinic

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Cancer Biology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Research Focus:gene regulatory network inference· 2 Organism:human· 2 Assay:bulk RNA-seq· 1 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:clustering· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Arvind PandayiD· 2 shared Farah Ramadan· 2 shared Neelima Yadav· 2 shared Annapoorna VenkatachalamiD· 1 shared Hugo VillanuevaiD· 1 shared Sadhana Dubey· 1 shared Sameer Bikram ShahiD· 1 shared Sameer Shah· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 35 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersCancer Biology

Arvind PandayORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

FANCM restrains structural genome evolution and defines a synthetic lethal dependency in BRCA1-deficient breast cancer

Farah Ramadan, Sameer Bikram Shah, Ujwal Subedi, Zachary D. Stephens, Annapoorna Venkatachalam, Neelima Yadav, Vickky Pandit, Sadhana Dubey, Sarah Latka, Hugo Villanueva, Scott H. Kaufmann, Arvind Panday

This study identifies FANCM as a synthetic lethal target in BRCA1-deficient breast cancer, showing it restricts structural genome instability and links replication stress to transcriptional changes.

bioRxiv · Sep 10, 2026 · PreprintCancer Biology

Comprehensive Analysis Reveals Adaptive DNA Repair and Replication Stress Networks in Genomically Unstable Breast Cancer

Farah Ramadan, Sara Zahraeifard, Neelima Yadav, Ujwal Subedi, Sameer Shah, Arvind Panday

The paper analyzes DNA repair and replication stress networks in genomically unstable breast cancer, identifying adaptive mechanisms that contribute to tumor resilience and therapeutic vulnerabilities.

bioRxiv · Aug 10, 2026 · PreprintCancer Biology