Author

W. C. Cole

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Physiology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Research Focus:cell signaling & trafficking· 1 Research Focus:cell type annotation· 1 Research Focus:development & morphogenesis· 1 Organism:human· 1 Assay:imaging· 1 Organism:mouse· 1 Organism:rat· 1

Frequent Co-Authors

J. A. MacDonald· 2 shared S. R. Turner· 2 shared C. Sutherland· 1 shared D. A. Carlson· 1 shared D. Belke· 1 shared M. Chappellaz· 1 shared M. Murali· 1 shared T. A. Haystead· 1 shared

Activity by Year

2020-2022 · peak 1/yr

Co-Author Collaboration Network

8 top collaborators · 20 mutual co-author links

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2 shared papersPhysiology

J. A. MacDonald

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2 papers

Recent·Most cited

Death-associated protein kinase 3 (DAPK3) regulates the myogenic reactivity of cerebral arterioles

S. R. Turner, D. A. Carlson, M. Chappellaz, C. Sutherland, T. A. Haystead, W. C. Cole, J. A. MacDonald

This study shows DAPK3 regulates actin cytoskeleton in cerebral arterioles, not Ca2+ sensitization, and its inhibition affects myogenic reactivity in rat and human vessels.

bioRxiv · Jun 10, 2022 · PreprintPhysiology

Enhanced diastolic dysfunction but preserved systolic function after acute pressure overload in the absence of smoothelin-like 1 protein

M. Murali, S. R. Turner, D. Belke, W. C. Cole, J. A. MacDonald

This study shows that SMTNL1 knockout mice exhibit enhanced diastolic dysfunction but preserved systolic function after acute pressure overload, resembling HFpEF pathophysiology.

bioRxiv · Oct 28, 2020 · PreprintPhysiology