Author

Y. Hovhannisyan

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

Click to filter author papers

Pathology· 2

Signature Assays, Organisms & Methods

Click to filter · or open tag

Approach:experimental assay· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Research Focus:host-pathogen & immune response· 1 Organism:human· 1 Organism:mouse· 1

Frequent Co-Authors

O. Agbulut· 2 shared Z. Li· 2 shared A. Behin· 1 shared A. Canette· 1 shared A. F. Klein· 1 shared A. Ferry· 1 shared A. L'honore· 1 shared A. Lilienbaum· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

2 shared papersPathology

O. Agbulut

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

Critical contribution of mitochondria in the development of cardiomyopathy linked to desmin mutation

Y. Hovhannisyan, Z. Li, D. Callon, R. Suspene, V. Batoumeni, A. Canette, J. Blanc, H. Hocini, C. Lefebvre, N. El-Jahrani, A. L'honore, E. Kordeli, P. Fornes, J.-P. Concordet, G. Tachdjian, , O. Agbulut

This study shows that mitochondrial defects in desmin-mutated cardiomyocytes contribute to cardiomyopathy and can be reversed by transferring normal mitochondria.

bioRxiv · Sep 14, 2023 · PreprintPathology

Desmin is a modifier of dystrophic muscle features in Mdx mice

A. Ferry, J. Messeant, A. Parlakian, M. Lemaitre, P. Roy, C. Delacroix, A. Lilienbaum, Y. Hovhannisyan, D. Furling, A. F. Klein, Z. Li, O. Agbulut

This study shows that desmin deletion in Mdx mice worsens muscle weakness, fragility, and fatigue resistance, while desmin gene transfer improves these features.

bioRxiv · Aug 23, 2019 · PreprintPathology