Author

Y. Mishina

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Cell Biology· 1 Immunology· 1

Signature Assays, Organisms & Methods

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Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Organism:mouse· 2 Assay:scRNA-seq· 2 Research Focus:cell-cell communication· 1 Research Focus:development & morphogenesis· 1 Approach:experimental assay· 1 Organism:human· 1

Frequent Co-Authors

A. James· 1 shared A. K. Huber· 1 shared B. Levi· 1 shared B. Li· 1 shared C. Hwang· 1 shared C. Pagani· 1 shared D. Nycz· 1 shared H. Chu· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperImmunology

A. James

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2 papers

Recent·Most cited

BMP signaling maintains auricular chondrocyte identity and prevents microtia development by inhibiting protein kinase A

R. Yang, H. Chu, H. Yue, Y. Mishina, Z. Zhang, H. Liu, B. Li

This study shows BMP signaling maintains auricular chondrocyte identity and prevents microtia by inhibiting PKA, through a switch from chondrogenic to osteogenic programs.

bioRxiv · Oct 3, 2023Cell Biology

Regulation of heterotopic ossification through local inflammatory monocytes in a mouse model of aberrant wound healing

A. K. Huber, M. Sorkin, C. Hwang, W. F. Carson, R. Menon, J. Li, K. Vasquez, C. Pagani, N. Patel, S. Li, N. Visser, Y. Niknafs, S. Loder, M. Scola, D. Nycz, , B. Levi

This study identifies TGFβ1-producing monocytes/macrophages as drivers of heterotopic ossification in a mouse model and shows CD47 activation can reduce HO by modulating these cells.

bioRxiv · Dec 11, 2019 · PreprintImmunology