Author

Y. Paung

Listed by name: these papers carry no ORCID for this author, so different people with the same name may be mixed together.

Biology Disciplines

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Biochemistry· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:drug response / drug discovery· 1 Assay:proteomics· 1 Assay:structural biology· 1

Frequent Co-Authors

A. Khan· 1 shared A. Lyczek· 1 shared A. M. Rangwala· 1 shared A. Weinheimer· 1 shared B. Ho· 1 shared B.-T. Berger· 1 shared H. Kim· 1 shared H. Philipose· 1 shared

Activity by Year

2021-2022 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 21 mutual co-author links

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1 shared paperBiochemistry

A. Khan

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2 papers

Recent·Most cited

Extended DNA binding interface beyond the canonical SAP domain contributes to SDE2 function at DNA replication forks

A. Weinheimer, Y. Paung, J. Rageul, A. Khan, B. Ho, M. Tong, S. Alphonse, M. Seeliger, H. Kim

This study identifies a conserved DNA binding motif in SDE2 that extends beyond the canonical SAP domain, enhancing ssDNA binding and replication fork stability.

bioRxiv · May 9, 2022 · PreprintBiochemistry

Mutation in Abl kinase with altered drug binding kinetics indicates a novel mechanism of imatinib resistance

A. Lyczek, B.-T. Berger, A. M. Rangwala, Y. Paung, J. Tom, H. Philipose, J. Guo, S. K. Albanese, M. B. Robers, S. Knapp, J. D. Chodera, M. A. Seeliger

The paper identifies a mutation in Abl kinase that causes imatinib resistance through altered drug dissociation kinetics rather than reduced binding affinity.

bioRxiv · Jun 28, 2021Biochemistry