CCRL2 promotes the interferon-γ signaling response in myeloid neoplasms with erythroid differentiation and mutated TP53
Nour Sabiha Naji, Sergiu Paşca, Theodora Chatzilygeroudi, Pablo Toledano-Sanz, Joseph Cataquiz Rimando, Yashvi Hemani, Brandy N. Perkins, Xinghan Zeng, C. Conover Talbot, Bogdan C. Paun, Abdulmuez Abdulmalik, Chen Lossos, Tatianna R. Boronina, Ilias Sinanidis, Panagiotis Tsakiroglou, , Theodoros Karantanos
This study identifies CCRL2 as a driver of IFN-γ signaling in TP53-mutated AML with erythroid differentiation, linking it to disease progression and poor outcomes.
bioRxiv · Feb 21, 2025 · PreprintCancer Biology