Author

Ilias Sinanidis

Johns Hopkins UniversityORCID 0009-0006-1112-3761Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Signature Assays, Organisms & Methods

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Assay:bulk RNA-seq· 2 Research Focus:cell type annotation· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:drug response / drug discovery· 1 Assay:proteomics· 1

Frequent Co-Authors

Bogdan C. Paun· 2 shared Panagiotis TsakiroglouiD· 2 shared Abdulmuez Abdulmalik· 1 shared Alexander Joseph AmbinderiD· 1 shared Amy Elizabeth DezerniD· 1 shared Benjamin G. Dubner· 1 shared Brandy N. Perkins· 1 shared C. Conover TalbotiD· 1 shared

Activity by Year

2025-2025 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersCancer Biology

Bogdan C. Paun

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2 papers

Recent·Most cited

Cancer BiologybioRxivPreprintMay 13, 2025

Serine auxotrophy is a targetable vulnerability driven by PSAT1 suppression in AML

Ilias Sinanidis, Panagiotis Tsakiroglou, Benjamin G. Dubner…·William Brian Dalton

The paper identifies serine auxotrophy in AML as a vulnerability driven by PSAT1 suppression, showing sensitivity to serine/glycine restriction and potential therapeutic targeting.

1 citation

humanbulk RNA-seq

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Cancer BiologybioRxivPreprintFeb 21, 2025

CCRL2 promotes the interferon-γ signaling response in myeloid neoplasms with erythroid differentiation and mutated TP53

Nour Sabiha Naji, Sergiu Paşca, Theodora Chatzilygeroudi…·Theodoros Karantanos

This study identifies CCRL2 as a driver of IFN-γ signaling in TP53-mutated AML with erythroid differentiation, linking it to disease progression and poor outcomes.

2 citations

humanproteomics

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