Author

Sherri M. Jones

University of Nebraska–LincolnORCID 0000-0001-5018-3659Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Neuroscience· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:mouse· 2

Frequent Co-Authors

Allison G. CurtisiD· 1 shared Andrianna I. AyiotisiD· 1 shared Antonia González-GarridoiD· 1 shared Charles C. Della SantinaiD· 1 shared Doris K. WuiD· 1 shared Gregg DuesteriD· 1 shared James Keller· 1 shared Jocelyn F. KreyiD· 1 shared

Activity by Year

2019-2023 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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1 shared paperNeuroscience

Allison G. CurtisORCID

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2 papers

Recent·Most cited

NeurosciencebioRxivPreprintFeb 28, 2023

Spontaneous allelic variant in Ush1g resulting in an expanded phenotype

Vladimir L. Vartanian, Jocelyn F. Krey, Paroma Chatterjee…·Peter G. Gillespie

The paper identifies a spontaneous mutation in the Ush1g gene in Neil1 knockout mice that causes an expanded phenotype including auditory and vestibular defects.

DOI 10.1101/2023.02.28.529432

mouseexperimental assay

𝕏 PostBluesky

NeurosciencebioRxivPreprintAug 6, 2019

Cytochrome P450 26b1-mediated specification of vestibular striola and central zones is required for transient responses in linear acceleration

Kazuya Ono, James Keller, Omar López…·Doris K. Wu

This study shows that Cyp26b1-mediated RA degradation during embryogenesis is essential for patterning vestibular zones, which are critical for transient responses to linear acceleration.

DOI 10.1101/726232

mouseexperimental assay

𝕏 PostBluesky