Mary J. Laws

This study shows that long-term exposure to a phthalate mixture in mice causes endometrial hyperplasia by enhancing estrogen and beta-catenin signaling pathways.

This study shows that phthalate monoesters alter membrane fluidity and disrupt cell-cell contacts in endometrial stromal cells.

The paper detects higher phthalate levels in adolescents' follicular fluid compared to oocyte donors and links these levels to changes in cumulus cell transcriptomes.