Author

Nandini Verma

Homi Bhabha National InstituteORCID 0000-0001-5260-8999Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Cancer Biology· 2

Signature Assays, Organisms & Methods

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Approach:experimental assay· 2 Organism:human· 2 Assay:proteomics· 2 Research Focus:cancer & tumor microenvironment· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1

Frequent Co-Authors

Anusha ShivashankariD· 2 shared Bhagya Shree ChoudharyiD· 2 shared Mohit Kumar JollyiD· 2 shared Nazia ChaudharyiD· 2 shared Sarthak SahooiD· 2 shared Sonal KhannaiD· 2 shared Subhakankha MannaiD· 2 shared Eeshrita Jog· 1 shared

Activity by Year

2023-2025 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 43 mutual co-author links

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2 shared papersCancer Biology

Anusha ShivashankarORCID

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2 papers

Recent·Most cited

Cancer BiologyCancer Cell InternationalPeer-reviewedFeb 19, 2025

EGFR-to-Src family tyrosine kinase switching in proliferating-DTP TNBC cells creates a hyperphosphorylation-dependent vulnerability to EGFR TKI

Nazia Chaudhary, Bhagya Shree Choudhary, Anusha Shivashankar…·Nandini Verma

This study identifies a hyper-phosphorylation state in drug-resistant TNBC cells caused by EGFR-to-Src kinase switching, making them vulnerable to EGFR TKI treatment.

9 citations

humanproteomics

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Cancer BiologybioRxivPreprintMay 18, 2023

GPX4-VIM equates a proliferating DTP state in TNBC subtypes with converged vulnerabilities to autophagy and glutathione inhibition.

Nazia Chaudhary, Bhagya Shree Choudhary, Sushmita Patra…·Nandini Verma

The paper identifies a proliferating DTP state in TNBC subtypes that is vulnerable to autophagy and glutathione inhibition, linking GPX4-VIM expression to survival outcomes.

DOI 10.1101/2023.05.18.541287

humanproteomics

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