Author

Benjamin R. Kroger

The University of Texas Southwestern Medical CenterORCID 0000-0001-5675-9102Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1

Signature Assays, Organisms & Methods

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Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:differential expression· 1 Assay:DNA methylation· 1 Research Focus:drug response / drug discovery· 1 Approach:experimental assay· 1

Frequent Co-Authors

Stephen S. ChungiD· 2 shared Toby ThomasiD· 2 shared Abishar Cherukuri· 1 shared Albert Son· 1 shared Alejandro MariñosiD· 1 shared Amy F. McLemoreiD· 1 shared Asmita MishraiD· 1 shared Bao Duy An Hoang· 1 shared

Activity by Year

2025-2026 · peak 1/yr

Co-Author Collaboration Network

10 top collaborators · 38 mutual co-author links

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2 shared papersCancer Biology

Stephen S. ChungORCID

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2 papers

Recent·Most cited

Persistent Malignant Stem Cells Drive Relapse After Transplantation in the Myelodysplastic Neoplasms

Benjamin R. Kroger, Roni Tamari, Carmelo Gurnari, Yuanyuan Ji, Yazan F. Madanat, Yi Huang, Reiko Tachibana, Toby Thomas, Albert Son, Peter Carlsgaard, Clayton W. Jackson, Vivian Irizarry-Gatell, Alejandro Mariños, Zachary Brady, Prapti Arvind Patel, , Stephen S. Chung

The paper detects malignant hematopoietic stem cells (HSCs) in myelodysplastic neoplasms (MDS) using low-input sequencing and shows they predict relapse after transplantation.

bioRxiv · Oct 29, 2026 · PreprintCancer Biology

Enigmatic missense mutations can cause disease via creation of de novo nuclear export signals

Michael McConville, Toby Thomas, Ryan L. Beckner, Benjamin R. Kroger, Catherine Valadez, Yuh Min Chook, Stephen S. Chung, G. P. Liszczak

The paper identifies missense mutations that create nuclear export signals, leading to disease, and shows therapeutic strategies can restore function.

Science Advances · Apr 16, 2025Peer-reviewedBiochemistry