Young‐Jin Son

This study shows that deleting PTEN enhances BRAF-driven regeneration of injured sensory axons across the DREZ, enabling long-distance growth into the spinal cord.

This study shows TEAD1 is essential for Schwann cell myelination, regeneration, and Remak bundle formation through mechanisms involving proliferation control and cholesterol biosynthesis.

The paper identifies oligodendrocyte precursor cells (OPCs) as the cells that stop sensory axons from regenerating into the spinal cord by forming synapses with them.

Genetic or HF-rTMS activation of B-RAF-MEK signaling promotes CST axon sprouting and functional regeneration after spinal cord injury.

This study shows that co-targeting B-RAF and PTEN in adult mice enhances dorsal column axon regeneration across and beyond spinal cord injury.

This study shows YAP/TAZ are necessary for Schwann cell remyelination but not proliferation after nerve injury, and their expression depends on axon regeneration.