Author

Ciro Menale

Federico II University HospitalORCID 0000-0001-5782-2089Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

Click to filter author papers

Cell Biology· 1 Physiology· 1

Signature Assays, Organisms & Methods

Click to filter · or open tag

Assay:bulk RNA-seq· 2 Research Focus:cell-cell communication· 2 Research Focus:differential expression· 2 Approach:experimental assay· 2 Organism:human· 2 Research Focus:cell type annotation· 1 Research Focus:gene regulatory network inference· 1 Assay:imaging· 1

Frequent Co-Authors

Anda Mihaela NaciuiD· 1 shared Andrea Ninni· 1 shared Andrea PalermoiD· 1 shared Anna CrescenziiD· 1 shared Cecilia BucciiD· 1 shared Cecilia Gentile· 1 shared Chiara Taffon· 1 shared Daniela Di GirolamoiD· 1 shared

Activity by Year

2026-2026 · peak 2/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

Click any collaborator node to inspect or filter shared papers

Focal author (center)Selected co-authorORCID co-authorUnlinked name

Curved chords = mutual co-authorship

1 shared paperPhysiology

Anda Mihaela NaciuORCID

Mutual co-authors in cluster

Author profile →

2 papers

Recent·Most cited

PhysiologybioRxivPreprintJul 16, 2026

Primary Hyperparathyroidism Reveals Limited Adipose Remodeling in Human

Andrea Palermo, Fabio Zaccaria, Andrea Ninni…·Daniele Lettieri‐Barbato

This paper investigates the effects of chronic PTH excess in PHPT on adipose tissue in humans and finds no significant thermogenic or energy-dissipating changes, but identifies a selective secretory phenotype.

DOI 10.64898/2026.07.05.736588

humanbulk RNA-seq

𝕏 PostBluesky

Cell BiologybioRxivPreprintMay 14, 2026

Motor Neuron Dysfunction in SORD Deficiency: Implications for Therapeutic Development in Peripheral Neuropathies

Giuseppina Divisato, Stefano Tozza, Emanuela Cascone…·Silvia Parisi

This study establishes an in vitro model of SORD deficiency using iPSCs and identifies mitochondrial dysfunction and oxidative stress as key mechanisms, suggesting AR inhibition as a therapeutic target for CMT2/SORD and diabetic neuropathies.

DOI 10.64898/2026.05.13.724849

humanbulk RNA-seq

𝕏 PostBluesky