Miloš Vittori

This study shows that MAGEA3/6 proteins in pancreatic cancer cells enhance metabolic flexibility by regulating glycolysis and mitochondrial metabolism, revealing therapeutic vulnerabilities.

This study investigates the impact of Magel2 loss on pituitary translation in a mouse model of PWS and SYS, revealing translatomic alterations and their functional implications.

This study compares mitochondrial ultrastructure and metabolism between glioblastoma stem-like cells and differentiated cells before and after temozolomide treatment, finding differences in mitochondrial morphology and metabolic dependence.