Author

Denis Štepihar

Texas Tech UniversityORCID 0009-0000-6875-4173Unclaimed ORCID profile · follow to get notified of new preprints

Biology Disciplines

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Biochemistry· 1 Cancer Biology· 1 Molecular Biology· 1

Signature Assays, Organisms & Methods

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Approach:experimental assay· 3 Research Focus:differential expression· 2 Organism:human· 2 Approach:statistical model· 2 Assay:bulk RNA-seq· 1 Research Focus:cell type annotation· 1 Research Focus:chromatin & epigenetic regulation· 1 Research Focus:database / resource· 1

Frequent Co-Authors

Klementina Fon TaceriD· 3 shared Maria Camila Hoyos Sanchez· 2 shared Miloš VittoriiD· 2 shared Tara BayatiD· 2 shared Andrey L. KaramysheviD· 1 shared Barbara Breznik Vittori· 1 shared Bing HaoiD· 1 shared Cristian Camilo Rodríguez Almonacid· 1 shared

Activity by Year

2026-2026 · peak 3/yr

Co-Author Collaboration Network

10 top collaborators · 29 mutual co-author links

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3 shared papersCancer Biology

Klementina Fon TacerORCID

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Author profile →

3 papers

Recent·Most cited

Cancer BiologybioRxivPreprintOct 21, 2026

Testis-Specific MAGEA3/6 Regulate Glycolysis to Promote Metabolic Flexibility and Reveal Therapeutic Vulnerabilities in Pancreatic Cancer Cells

Sima Tozandehjani, Vesna Jurjevič, Barbara Breznik Vittori…·Klementina Fon Tacer

This study shows that MAGEA3/6 proteins in pancreatic cancer cells enhance metabolic flexibility by regulating glycolysis and mitochondrial metabolism, revealing therapeutic vulnerabilities.

DOI 10.64898/2026.10.20.760599

humanproteomics

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Molecular BiologybioRxivPreprintMay 14, 2026

Loss of MAGEL2 Disrupts Pituitary Translation in a Mouse Model of PWS and Schaaf-Yang Syndrome

Tara Bayat, Maria Camila Hoyos Sanchez, Cristian Camilo Rodríguez Almonacid…·Klementina Fon Tacer

This study investigates the impact of Magel2 loss on pituitary translation in a mouse model of PWS and SYS, revealing translatomic alterations and their functional implications.

DOI 10.64898/2026.05.12.724462

mousebulk RNA-seq

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BiochemistrybioRxivPreprintApr 28, 2026

Mechanisms of USP7/MAGEL2 Complex Assembly and Its Mutational Disruption in Neurodevelopmental Diseases

Emilie J. Korchak, Gabriela Soriano, Irina Semenova…·Irina Bezsonova

This study investigates the assembly mechanism of the USP7/MAGEL2 complex and identifies mutations that disrupt its formation, linking it to neurodevelopmental diseases.

1 citation

humanprotein structure

𝕏 PostBluesky